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SUKKUR:The National Accountability Bureau (NAB) Sukkur arrested on Tuesday two government officials on charges of misappropriating pensioners' funds to the tune of more than Rs150 million.The accused were arrested from the Sindh High Court (SHC) premises in Sukkur after the court rejected their pre-arrest <a href=https://www.adidas-samba-adidas.it>adidas samba nera</a> bail.According to NAB Sukkur spokesperson, the bureau received complaints against Muhammad Arif Teevno, former branch manager of National Bank of Pakistan's (NBP) Bhiria City branch in Naushero Feroz, Muhammad Ali Panhwar, former district accounts officer in Naushero Feroz, Nizamuddin Solangi, former sub-accountant of district accounts o <a href=https://www.adidascampus.com.de>adidas campus 80s</a> ffice in Naushero Feroz, Ghulam Asghar Rajpar, an agent, and others regarding embezzlement of government funds and misuse of authority.Embezzlement Case: NAB arrests four Sindh govt officials聽聽The spokesperson further said around 63 accused, including the cash <a href=https://www.nbbalance.fr>newbalance 550</a> ier of NBP Bhiria City branch, Ahmed Khan Solangi, and former medical superintendent of Kamal Children National Hospital experts developed a new pre-clinical gene therapy for a rare disorder, known as limb-girdle muscular dystrophy (LGMD) 2B, that addresses the primary cellular deficit associated with this d <a href=https://www.nbbalance.de>new balance 990</a> isease. Using a single injectio <a href=https://www.jordann.at>jordan2</a> n of a low dose gene therapy vector, researchers restored the ability of injured muscle fibers to repair in a way that reduced muscle degeneration and enhanced the functioning of the diseased muscle. The treatme <a href=https://www.dunks.fr>nike dunk high</a> nt was safe, attenuated fibro-fatty muscle degeneration, and restored myofiber size and muscle strength, according to thestudypublished in theJournal of Clinical Investigation.With an incidence of less than 1 in 100,000, LGMD2B is a rare disorder caused by a genetic mutation in a large gene called dysferlin. This faulty gene leads to muscle weakness in the arms, legs, shoulder and pelvic girdle. Affected children and adults face trouble walking, climbing stairs, and getting out of chairs. Individuals typically lose the ability to walk

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